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Reduce Colorectal Cancer Risk

From eNewsletter 10/5/2020

DID YOU KNOW that in a study from last week's Gut, researchers listed several nutrients that can reduce colorectal cancer and mortality risk? Magnesium, folate, selenium, carotenoids, along with a high consumption of fruit and vegetables, and fiber were mentioned as having the strongest effect on lowering colorectal cancer risk. PURE ENCAPSULATIONS ESTER-C & FLAVONOIDS, NOW CALLED ESSENTIAL-C & FLAVONOIDS, ARE BACK! Ester-C & Flavonoids, now called Essential-C & Flavonoids, is finally available. The formula contains buffered vitamin C with a small amount of quercetin and rutin. VIRUS PREVENTION

Continue with extra immune support until at least summer of 2021. SARS-CoV-2 knows no boundaries and does not discriminate. Besides, SARS-CoV-2 is not the only virus we fight. There is influenza (flu), norovirus (stomach flu), adenovirus (common cold), and four other coronaviruses (common cold), among others. Prevent and Fight Coronavirus 2.0 is our must-read protocol. For detailed advice about conventional, as well as integrative treatments, read Steve Minsky's COVID-19 Condition Monograph.


Steve and Bonnie: What really ignites the breakdown of stored fat molecules are nerves embedded in the fat tissue. A new study in Nature reveals that if these fat-burning neurons receive the right signal, they have an astonishing capacity to grow, which is good.

The signal is the hormone leptin, which is released by the fat cells themselves. Researchers found that the normally bushy network of neural fibers within fat tissue shrinks in the absence of leptin, limiting the ability to burn the energy stored in fat. Leptin relays signals between fat deposits and the brain, allowing the nervous system to curb appetite and boost energy expenditure to regulate body weight. When mice are genetically engineered to stop producing leptin, they grow three times heavier than normal mice. When mice are given a dose of leptin, however, they quickly begin to eat less and move more. But when the researchers treated them longer, more profound changes occurred: the animals started to break down white fat, which stores unused calories, at normal levels, and regained the ability to use another form of fat tissue, brown fat, to generate heat. Unfortunately, most obese people produce high levels of leptin, and show a diminished response to hormone injections, suggesting that their brains are resistant to the hormone. Thus, the researchers are going to look for ways to bypass leptin resistance by directly activating the nerves in fat cells. This could have a profound benefit for leptin-resistant persons. Another strategy to address the difficulty of reducing excess fat tissue is to look at the genetic piece. We have cited a plethora of studies showing how inextricably linked genes are with weight. In a study from last month's Genome Research, researchers showed how important proper fatty acid balance is for fat tissue breakdown. Specifically, they found mutations of the FADS gene were linked to overweight and obesity. FADS is a gene we test in our Pure Genomics screening. FADS has to do with how well we turn oils into beneficial omega-3 fatty acids. When we do not do this properly, we create an overabundance of omega-6 fatty acids, which are often inflammatory. Inflammation impairs fat burning. Of course, the therapy to address a FADS mutation is to eat fatty fish or take a fish oil supplement.


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