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CoQ10's Critical Role

From eNewsletter 11/2/2020

DID YOU KNOW that a study from Molecular Metabolism found that vitamin A plays an important role in the function of adipose (fat) tissue and affects global energy metabolism? You've heard that cold temperatures can increase energy consumption by turning white fat into brown fat, which generates heat. The researchers discovered that increased vitamin A levels leads to this transformation. Thus, vitamin A is critical for helping convert "bad" white adipose tissue into "good" brown adipose tissue to stimulate fat burning and heat generation. That said, nobody should run out and start taking megadoses of vitamin A. Understanding how well you genetically convert beta carotene into vitamin A is priority number one. Once understood, we can then assess if, and how much, vitamin A you require. VIRUS PREVENTION Continue with extra immune support until at least summer of 2021. SARS-CoV-2 knows no boundaries and does not discriminate. Besides, SARS-CoV-2 is not the only virus we fight. There is influenza (flu), norovirus (stomach flu), adenovirus (common cold), and four other coronaviruses (common cold), among others. Prevent and Fight Coronavirus 2.0 is our must-read protocol. For detailed advice about conventional, as well as integrative treatments, read Steve Minsky's COVID-19 Condition Monograph.

Have a happy, healthy day! Steve and Bonnie


Steve and Bonnie: Co-Enzyme Q10 (CoQ10) supplementation may be behind improvements in symptoms seen in disorders like heart disease, diabetes and kidney disease, according to a review in Nutrients.

Researchers indicate the molecule's ability to influence fatty acid metabolism, as well as mediating gene expression, including those involved in inflammation. CoQ10 has a central role in the metabolism of all cells, and a CoQ10 deficiency is linked to the pathogenesis of a range of disorders. Brain, muscle and kidney tissues are particularly susceptible to the metabolic consequences of a deficit in the status of CoQ10. Early identification of such primary CoQ10 deficiencies is essential, since some may show remarkable clinical improvement following CoQ10 supplementation when administered at an early stage of disease. CoQ10 is not a vitamin, since it is produced by various tissues within the human body serving to protect cellular membranes and both mitochondrial and extra-mitochondrial structures from free radical induced oxidative stress. CoQ10 is also involved in vitamin C and E regeneration and has a role as a mediator of inflammation and cholesterol metabolism. A small amount of CoQ10 (approximately 5 mg) is obtained from animal protein, with most of the daily requirement being synthesized within the body mainly in the kidney, heart, skeletal muscle and liver. Recent CoQ10 studies include the Q-SYMBIO 2 trial that found cardiac-related deaths and deaths from all causes fell by 43% and 42% respectively among patients taking 300mg of CoQ10 daily day along with their conventional medication over a two-year period. Type 2 diabetes patients also benefit from CoQ10 supplementation as patients supplementing with 200mg of CoQ10 per day for three months were found to have significantly reduced levels of glycated hemoglobin (HbA1C). Another study which saw type 2 diabetics taking 150mg of CoQ10 a day found significantly improved blood glucose and HbA1C levels after three months. Quality is paramount for supplemental CoQ10 because in the absence of crystal dispersion in supplemental CoQ10 formulations, bioavailability could be reduced by 75 percent. Providing the proper method for creating bioavailable CoQ10 is why high quality CoQ10 can be expensive.


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